What happens to blood flow during Reactive Hyperemia?

During reactive hyperemia, blood flow to a specific area temporarily increases after the release of pressure that had previously occluded blood flow. This phenomenon occurs as a physiological response to ensure that the tissues receive an adequate supply of oxygen and nutrients following a period of reduced blood flow.

When an external pressure or blockage is applied, such as during a blood pressure cuff application, blood flow to that region is restricted or entirely cut off. Once the pressure is released, the blood vessels dilate and blood rushes back into the area, resulting in a transient increase in blood flow. This response is a crucial mechanism that helps the body restore normal function and prevent tissue damage.

The other options do not accurately describe the process. Blood flow does not remain constant or decrease significantly in the context of reactive hyperemia; rather, it is the lack of flow during the occlusion that triggers the response. Likewise, blood flow does not cease completely but instead is temporarily halted until the release of pressure. Understanding this response is essential in vascular physiology and has implications in clinical settings for assessing blood flow and vascular health.